Dihydroquercetin as a means of preventative

Very often I hear the statement that his grandfather, father, mother sister suffered from this or that. There’s a lot of truth. Through our genes we inherit all the baggage of our ancestors. Often we not even knowing that we are in the risk group.
Group risk does not mean, however, that we will be sick but that we are prone to this disorder. We can only help him to our way of life and conscious choice.
Make individual surveys on our health we can do at home, answering a few questions: who in the family was ill, or suffering from still if this is disorder classified as lifestyle diseases (list below), or died. Useful will also be talk with loved ones, and watch.
If the answer is affirmative then you are probably at group of risk.

Mostly we hear about diabetes mellitus, cardiovascular disease (atherosclerosis, heart attack, stroke, high cholesterol, hypertension), cancer, depression, problems with the eyes and skin). Not surprisingly, according to the World Health Organization, every year due to diseases of civilization are dying more than 17.3 million people.

In the development of “Genetic determinants of cardiovascular disease” dr hab. n. med. Alice Bortkiewicz and Marcin Kosobudzki move considerations for the role of genes in the circulatory system. Genetic testing in diseases of the circulatory system are not commonly used and still remain at the stage of research. Some results will probably never find practical application. It seems, however, that with the development of these studies (e.g. genes predisposing to the onset of hypertension or coronary heart disease) to make progress in the prevention and treatment of cardiovascular disease.

Determination of suitability to the onset of the disease will help to create dyspansery groups in order to monitor the State of health of persons at risk, which in turn would reduce the death rate from the disease. It turns out that a combination of genetic factors and the environment determines the clinical picture and the course of coronary heart disease (ChNS). Genes associated with lipid metabolism genes are apolipoprotein B and apolipoprotein E genes, lipoprotein lipase gene, and gene protein which is responsible for esters transport. It is believed that the high risk of premature of ChNS and heart attack in people at a young age (before 45. rż.) affects the presence of variant of the ApoE gene, E4. This option is also responsible for the increased risk of death due to heart attack and ChNS.From the article, we can also learn that the genes responsible for metabolism and action of sex hormones may have a different impact on the development of coronary heart disease in men and women. In addition, hypertension occurs in about 20% of the population. The etiology of this disease is complex and multifactorial. Of great importance for pathogenesis of hypertension have genetic testing. About 40% of the variation of the pressure in the population is subject to the action of genes. Pathogenesis of hypertension is mainly multigenic. Effects of environmental factors result in disclosure to the phenotypic characteristics of the disease, which is the increase in blood pressure. ACE gene may play a large role in the predisposition to hypertension.

The authors clearly indicate that environmental factors-stress, nutrition, smoking, alcohol consumption, excess weight determine the development of the disease in our body and responsible for this state of affairs are oxygen radicals.

An important cause of atherosclerosis is a welcoming family-run genetic load. It is the disadvantage of the hypercholesterolemia pathologically excessive levels of bad cholesterol in the blood. This genetic disease in its heaviest form occurs very rarely, however, its lighter form is relatively frequent, because 1 500 children!. The disease is characterized by a deficiency of the so-called. receptors necessary for removal from the body of “bad” LDL cholesterol.

Therefore, patients have a significantly elevated levels of “bad” LDL cholesterol in the blood serum. In people affected by the severe form of this disease leads to the development of atherosclerosis in childhood. On the occurrence of hypercholesterolemia control measurement indicates the family of cholesterol in the blood, as well as in the case of negligence of research-yellow skin on the eyelids. Study of genetic effects of carrying a family type e4 allele of hypercholesterolemia indicate exponentiation pathology through the influence of the traditional risk factors for atherosclerosis, such as cigarette smoking, elevated levels of blood fat (triglycerides)
Internet forums are full of cholesterol test results, LDL and HDL and tri glycerides. The results are alarming because the schema is as follows: male 37 years, goes to work by car, a lot of sitting, on every customer drinks coffee, he returns to the house, is so tired, so he does not have the strength to exercise, sleeps 5 hours, for dinner ate delicious wings with boxes and no vegetables.

Knowing about this, that we are exposed to an instance of a given disease unit we should consciously begin to identify what stage we are (among other things through the study focused on the unit and use the treatment on the basis of antioxidants). With the help of antioxidants come. There are many compounds kemferol, naringenina, quercetin, luteolin. However, it is recognised that Dihydroquercetin considered the strongest antioxidant. Its antiradical activity is disclosed in concentrations of 10 ⁻ ⁴-10 ⁻ ⁵%. Because of its antioxidant action affects the strengthening blood vessels, which may be genetically weakened and by external factors (free radicals) that are delivered to our body in multiple ways.
In the event of a disease entity of one of the member of the family should be included in prevention whole family. Model- person.(sick) + 3persons (risk group).
I refer to the research-medical portals out there you can find more information about heart disease and the risk of complications of atherosclerosis at an early age.

1/The genetic determinants of cardiovascular diseases, dr HAB. n. med. Alice Bortkiewicz, Marcin Kosobudzki Institute of occupational medicine to them. Prof. Dr. med. Jerzy Eurohealthnet – the Plant Physiology and Ergonomics ul. St. John the Baptist. Thérèse of the child Jesus, 8, 91-348 San Jose family medicine Forum 2012, vol. 6, no. 1, 1-13 http://www.czasopisma.viamedica.pl/fmr/article/download/17939/14140

Leave a Reply

Your email address will not be published. Required fields are marked *