Cells function of our body depends on a continuous supply of oxygen from the blood. If the blood supply to a tissue would cease, the situation becomes very serious. After a few or a several minutes of cut-off of oxygen supply (depending on the type of tissue) in the tissue are irreversible changes. Many studies of this type was performed on the heart muscle. They showed that few minute cutting off blood supply to the heart muscle causing arrhythmia, which persists spontaneously after a certain time. Longer, few or ten minutes ischemia results in an characteristic set of changes, known as the “stunned heart” or systolic heart disorders of abnormal contractions of the heart, which are not accompanied by myocardial necrosis. These changes will go away after a few-dozen days. Ischemia (part) of the heart muscle lasting longer than several minutes causes it to die off.
Heart arrhythmias can lead to serious and irreversible changes in the body. The use of Dihydroquercetin followed by improvement of microcirculation and contractility of the myocardium, coronary blood flow, and decrease the viscosity of the blood.
Atherosclerosis is a serious problem developed societies, where every other person dies as a result of a heart attack or stroke caused by a sudden inhibition of blood flow in the vessels, narrowed as a result of internal membranes from swelling of blood vessels. There are three types of such lumps: oclusions fatty -slightly protruding inside the oblong dish, the yellow areas, from which arise the plaque atherosclerotic-oval, white, more protruding structure with a diameter of about 1 cm, which can stop a light blood vessel. Suction disorders the bloodstream as a result of failure of oxygen cell suffocated and starves, which leads to the disease and destroy cells. Heart attack or stroke occurs when a atherosclerotic plate completely closes the flow of blood through the vessel (usually as a result of the creation of the scute clot) cutting off the blood supply to the heart or brain.
The modern concept concerning on the mechanism of the formation of atherosclerotic deposits in the blood vessels, is the result of intensive research of the last decade, assumes that atherosclerotic plaques are created by cells filled with lipids than from damaged by reactive oxygen species plasma lipoproteins. The activity of the tube-capillary-protecting taxifolin prevents the destruction of cell membranes, tissue, strengthens the walls of blood vessels and capillaries, stimulates and reconstruction: lighten capillaries, enhances the blood flow, because cells begin a stage of renewal.
Taxifolin is designed to prevent the next attack by protecting the entire network of tiny blood vessels. As a curiosity, I can provide that the total length of all the blood vessels in the adult human being is more than 160 thousand. miles! It’s four times more than the length of the equator. In a child – this is almost 100 thousand. miles. And therefore the body on a daily basis takes the fight against reactive metabolites form of oxygen that cause destruction of so huge amount of capillaries.