The reactive oxygen species are considered to be the factor pathogenetic heart dysfunction induced by ischemia and in the development of congestive arrest, what was confirmed in 2000. Ide and partners were making direct measurement of increased generation of radical system in inefficient heart.
It has been shown that to increased generation of free radicals occur during myocardial infarction and in the development of cardiomyopathy. Research of the last decade show that reactive oxygen species can induce arrhythmia and that increased production of reactive oxygen species occurs during cardio-surgical treatments. Free radical reactions seem also to play a key role in recoil after a heart transplant.
One of the key elements affecting the correct functioning of the heart beat are the capillaries (the capillary) which is supplied oxygen and nutrients to the cells.
Suction disorders the bloodstream as a result of failure of oxygen cell suffocated and starves, which leads to the disease and destroy cells. Professor Sylwester Terek, recommended that you provide the body with antioxidants trapping free radicals.
Atherosclerosis is a serious problem developed societies, where every other person dies as a result of a heart attack or stroke caused by a sudden inhibition of blood flow in the vessels, narrowed as a result of internal membranes from swelling of blood vessels. There are three types of such lumps: oclusions fatty -slightly protruding inside the oblong dish, the yellow areas, from which arise the plaque atherosclerotic-oval, white, more protruding structure with a diameter of about 1 cm, which can stop a light blood vessel. Suction disorders the bloodstream as a result of failure of oxygen cell suffocated and starves, which leads to the disease and destroy cells. Heart attack or stroke occurs when a atherosclerotic plate completely closes the flow of blood through the vessel (usually as a result of the creation of the scute clot) cutting off the blood supply to the heart or brain.
The modern concept concerning on the mechanism of the formation of atherosclerotic deposits in the blood vessels, is the result of intensive research of the last decade, assumes that atherosclerotic plaques are created by cells filled with lipids than from damaged by reactive oxygen species plasma lipoproteins. The activity of the tube-capillary-protecting taxifolin prevents the destruction of cell membranes, tissue, strengthens the walls of blood vessels and capillaries, stimulates and reconstruction: lighten capillaries, enhances the blood flow, because cells begin a stage of renewal.
Taxifolin is designed to prevent the next attack by protecting the entire network of tiny blood vessels. As a curiosity, I can provide that the total length of all the blood vessels in the adult human being is more than 160 thousand. miles! It’s four times more than the length of the equator. In a child – this is almost 100 thousand. miles. And therefore the body on a daily basis takes the fight against reactive metabolites form of oxygen that cause destruction of so huge amount of capillaries.