Taxifolin (taxifolin, dihydroquercetin, diquertin) in combination with ascorbic acid (vitamin C) impairs cerebral cortex damage caused by ischemia of the brain. Studies have been carried out using electron microscopy and electrophysiology research on laboratory rats. The research was conducted in the Russian Academy of Sciences Research Center in Tomsk, Institute of pharmacy.
A link to the page of the Institute: http://www.pharmso.ru/english.html
Featured article was translated into English from “Byulleten, Eksperimental’noi Bilogii and Meditsiny, Vol. 130, No. 11, pp. 543-547, November 2000.
See original article:
Cerebroprotective effects of Diquertin and ascorbic acid.
Researches have shown that repeated administration of dihydroquercetin (taxifolin) weakens the destruction caused in the cerebral cortex by brain ischemia.The test was very drastic. Taken 28 rats, they forthe first ligation of the carotid artery so as to reduce the flow of blood by half, then then treated with taxifolin. Five days later they were sacrificed and their brains were examined. The text is very scientific, and I’m not an expert, so I can’t even understand what in fact were studied. I’m not a doctor. The most important is conclusion, however, is that in conjunction taxifolin with vit-C reduces damage in cerebral cortex induced by ischemia. Probably is hard to find among your friends the case of a patient who had such ischemia and taking taxifolin, because how to examine whether this taxifolin help? How to survive, it raises the question whether if he did not take it too to survive? Maybe someone has a research on people that would confirm this action of taxifolin.
Atherosclerosis is a serious problem developed societies, where every other person dies as a result of a heart attack or stroke caused by a sudden inhibition of blood flow in the vessels, narrowed as a result of internal membranes from swelling of blood vessels. There are three types of such lumps: oclusions fatty -slightly protruding inside the oblong dish, the yellow areas, from which arise the plaque atherosclerotic-oval, white, more protruding structure with a diameter of about 1 cm, which can stop a light blood vessel. Suction disorders the bloodstream as a result of failure of oxygen cell suffocated and starves, which leads to the disease and destroy cells. Heart attack or stroke occurs when a atherosclerotic plate completely closes the flow of blood through the vessel (usually as a result of the creation of the scute clot) cutting off the blood supply to the heart or brain.
The modern concept concerning on the mechanism of the formation of atherosclerotic deposits in the blood vessels, is the result of intensive research of the last decade, assumes that atherosclerotic plaques are created by cells filled with lipids than from damaged by reactive oxygen species plasma lipoproteins. The activity of the tube-capillary-protecting taxifolin prevents the destruction of cell membranes, tissue, strengthens the walls of blood vessels and capillaries, stimulates and reconstruction: lighten capillaries, enhances the blood flow, because cells begin a stage of renewal.
Taxifolin is designed to prevent the next attack by protecting the entire network of tiny blood vessels. As a curiosity, I can provide that the total length of all the blood vessels in the adult human being is more than 160 thousand. miles! It’s four times more than the length of the equator. In a child – this is almost 100 thousand. miles. And therefore the body on a daily basis takes the fight against reactive metabolites form of oxygen that cause destruction of so huge amount of capillaries.